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37,38'53 The crossbridges in which the motor domain is not at the rigor position on actin and the lever arm is up at a high angle may represent weak contacts between myosin and actin at the beginning of a power stroke that realign on actin to reach a strong binding position. Or, they may be nonstereospecific contacts that do not evolve into force producing interactions. The range of rebuilt SI models in HST was ordered into a hypothetical sequence compatible with a continuously attached, progressive -12 nm power stroke comprising an angular range of > 100° to 45°.
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EMBO J 1999; 18:1793-1804. 46. Moerman DG, Plurad S, Waterston RH. Mutation in the unc-54 myosin heavy chain gene of Caenorhabditis elegans that alter contractility but not muscle structure. Cell 1982; 29:773-781. 47. Epstein HF, Bernstein SI. Genetic approaches to understanding muscle development. Dev Biol 1992; 154:231-244. 48. Ferrus A, Acebes A, Marin MC et al. A genetic approach to detect muscle protein interactions in vivo. Trends Cardiovasc Med 2000; 10:293-298. 49. Homyk Jr T, Emerson Jr CP.